Eating a high carb and high sugar diet does not promote diabetes.

Eating a low carb diet does not reverse or prevent diabetes.

Shocking, I know. Here me out. The common and popular understanding about what is going on isn't quite right.

For the purposes of this discussion, I will define diabetes as an impairment to your ability to rapidly and effectively process blood glucose, should glucose happen to enter your blood stream.

You might have diabetes even if you eat a zero-carb diet. You wouldn't know. Because without eating sugars you can't tell if your body is good at processing those sugars. All the nasty symptoms and damage caused by diabetes will be absent while you don't eat sugars, which is great. But once you start eating sugars again you could find that your body cannot tolerate them.

Diabetes is caused by two things: (1) visceral fat that has invaded your pancreas, and (2) beta cells that are not very fat tolerant (this is genetically determined). If your pancreatic beta cells are not very fat tolerant, and you have a tendency to store fat viscerally, then you will become diabetic at perhaps a low body weight, maybe even at 25 BMI. You drew the short straw. Some people have very tolerant beta cells and store fat subcutaneously and can get up to massive BMIs without becoming diabetic. Everybody has a personal body weight over which they will become diabetic that is mostly genetically determined and not known to be changeable.

What happens with diabetes is that as the pancreas becomes more and more fatty, the beta cells can't tolerate the fattyness of the environment and stop behaving like beta cells. They don't die, they are still there, but they cease operation.

The good news is that if you lose weight, those beta cells will resume operation and your diabetes will be cured.

So going back to my initial statements, it isn't about carbs or fat or protein in your food. It is about total energy consumption, total body weight. Becuase your body converts excess carbs into fat, and it converts excess protein into fat too. And if you eat in excess, eventually your pancreas will become too fatty for your beta cells.

People think sugars/carbs cause diabetes probably because diabetics can't tolerate sugars. But carbs only cause diabetes if you eat them to excess, causing you to become overweight and your pancreas to become fatty. You can do this to yourself by overeating protein too.

People think a low-carb diet cures diabetes, but this is only if that diet causes you to lose weight below your diabetic threshold. It also happens to mask diabetes since you aren't eating sugars so you really can't tell if your beta cells are working or not while you are on the diet.

This post was informed by research done at Newcastle University.

Type 1 diabetes is caused by failure of the pancreas to produce insulin, typically caused by destruction of beta cells by the immune system. This is not the kind that’s caused by diet.

Type 2 diabetes is caused by (or defined as) insulin resistance, meaning that the cells of your body stop responding properly to insulin. It’s not primarily caused by dysfunction of the pancreas, although there can be secondary pathological effects on the pancreas. Diet is a big contributing factor. If someone is well fed all the time, then the insulin levels in the blood are always high and never drop to zero. The fact that cells become insulin resistant could be related to the fact they have been swimming in high levels of insulin for as long as they can remember. Fasting will drop your insulin level to basically zero after maybe 18-24 hours (or longer depending on activity level). If your cells have forgotten what it feels like to exist without high levels of insulin around, that’s probably not a good thing, and fasting / decreasing carbs in your diet will probably improve insulin resistance.

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Discussion

Yes I didn't make myself clear. I am referring only to Type 2.

Insulin resistance by your cells which are overfed while you still have more food for them is a core part of the pathology. I would have defined Type 2 like you did, based on insulin resistance, before I read the book I'm reading "Living without Diabetes", the COUNTERPOINT study and related work (counterpoint reflections, counterbalance), the DiRECT study, etc.

So the theory that seems to have borne out is called the "twin-cycles theory":

First of the twin cycles - the vicious cycle in the liver. An extra mouthful of food daily over a long period of time causes fat in the liver to build up. This makes the liver unable to respond properly to insulin. Blood glucose starts to creep up and this causes higher background levels of insulin. This insulin then oils the wheels for glucose to be turned into liver fat.

Second of the twin cycles - the vicious cycle in the pancreas. The normal fat export from the liver can deal with the high liver fat - until the safe storage depot under the skin is full. Then blood levels of fat rise. The pancreas is one of the tissues susceptible to the increased fat. Eventually, after several years, this causes the insulin producing cells to shut down. Over a short time, blood levels then rise.

Some people genetically have tissues that are more insulin resistant from birth. These people are more susceptible. But cellular insulin resistance is mainly a reaction to a state of being over-fed. It can change fairly quickly once you stop eating. Whereas the pancreatic switch into diabetes needs a more drastic dieting to overcome.

I am not a doctor, do not consider this to be medical advice.