No, no, no. That's not the hypothesis, that's not how prions work, and it doesn't make any sense.

The turbocancer hypothesis centers around incorporation of an amplification sequence (via retrotransposon) into the human genome.

Prions are misfolded proteins that induce a chain reaction inducing normal proteins to also misfold, thereby causing neurodegenerative disease of the central nervous system. The brain controls nerves that go to all parts of the body so it looks like the disease "spreads everywhere", but the prions themselves are not thought to.

Creative thinking is great but you can't just make up a new disease and new pathophysiology out of thin air. That's just as assinine as "flatten the curve."