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The threat here is not that far off - which is why it is of grave importance that we push for an explanatory theory of long COVID (& post-acute infection syndrome) that can fully account for the totality of symptoms observed after an initial infection with SARS-CoV-2 despite no clinical findings of active infection

ASIDE: see my earlier post re: human psychology & deferred risk / risk aversion:

https://mastodon.social/@persagen/110707507862338608

Why people struggle to understand climate risk

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THE SCIENCE BEHIND LONG COVID

Multiple studies published in the journal Nature Communications (one published last year and one published in February of this year) explain how COVID-19 has the ability to trigger the aggregation of proteins within the human body.

The research suggests that SARS-CoV-2 can cause normal proteins to abnormally misfold.

These misfolded proteins are known as "amyloids," which are toxic to cells when they build up.

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